50多岁的男性可能因有毒“永久化学物质”而加速衰老。
Men in their 50s may be aging faster due to toxic 'forever chemicals'

原始链接: https://www.cnn.com/2026/02/26/health/forever-chemicals-aging-men-wellness

## PFAS 与加速衰老:一项新研究 最近发表在《衰老前沿》上的一项研究表明,接触 PFAS(几乎存在于所有美国人身上的“永久化学物质”)可能会加速生物衰老,尤其是在 50-65 岁的男性中。研究人员分析了超过 300 名成年人的数据,发现血液中较高的 PFAS 水平与表观遗传衰老程度增加有关——这是衡量生物年龄与实际年龄的指标。 这种影响在中年男性中最为明显,这可能是由于 PFAS 扰乱内分泌系统,影响睾酮水平和生殖健康。虽然在女性中也观察到一些关联,但关联性较弱。该研究还强调了对较少研究的 PFAS 化学物质 PFNA 和 PFOSA 的潜在担忧。 研究人员强调,这项研究表明的是*关联性*,而非直接的*因果关系*,但它强化了进一步调查的必要性。建议通过净水器和谨慎消费处理过的材料来减少接触,然而,显著降低风险需要更广泛的监管行动和环境清理工作。

发表在《衰老前沿》上的一项最新研究表明,50多岁的男性可能因暴露于全氟/多氟烷基物质(PFAS,“永久化学物质”),特别是PFNA和PFSA而经历加速衰老。该研究强调了与这些新兴污染物相关的性别和年龄特定风险,以及它们对表观遗传衰老的影响。 Hacker News上的讨论指出,PFAS广泛存在于日常产品中,例如滑雪/单板蜡(直到最近才使用)和含有聚四氟乙烯(PTFE)的“永久”烘焙纸。人们对PTFE在相对较低温度(约260°C)下分解表示担忧,尽管它通常被认为是正常烹饪安全的。 总体情绪强调了PFAS的问题性——从其制造过程到最终降解——以及避免其生产的愿望。
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原文

The “forever chemicals” known as PFAS appear to be aging men faster in their 50s and early 60s, a new study found.

Called forever chemicals due to the years it takes for them to break down, perfluoroalkyl and polyfluoroalkyl substances are present in the blood of an estimated 98% of Americans, according to the National Academies of Sciences, Engineering, and Medicine.

Epigenetic aging — a measurement of one’s biological age instead of chronological age — was most advanced in men, according to the study.

“The associations between PFAS exposure and accelerated epigenetic aging were strongest in men aged 50 – 65,” said senior study author Xiangwei Li, a professor of epidemiology at China’s Shanghai Jiao Tong University School of Medicine, in an email.

“In younger men and those over 65, the associations were weaker and generally not statistically significant,” Li said. “We observed some associations in women, but they were generally smaller and less consistent than those seen in middle-aged men.”

The findings indicate a “sex-specific effect” that can be expected for chemicals that disrupt the endocrine system, a vital network that regulates key bodily functions such as growth, metabolism, mood and reproduction, said Jane Muncke, managing director and chief scientific officer at the Food Packaging Forum, who was not involved in the study.

The forum is a nonprofit foundation based in Zurich, Switzerland, that focuses on science communication and research on plastics and other chemicals used in industry.

In men, the accumulation of PFAS may lower testosterone levels, impair sperm quality and increase risks of testicular and kidney cancers.

Earlier studies show women appear to eliminate certain PFAS faster than men due to pregnancy, breastfeeding and menstrual blood loss. Studies have also found the difference in PFAS accumulation between women and men narrows after menopause.

While the new study’s results are interesting, they “cannot be interpreted as cause-and-effect but rather are puzzle pieces, or building blocks, to establish biological plausibility,” Muncke said in an email.

The American Chemistry Council, which represents the industry, told CNN the “exploratory” study was based on a small sample of older adults using data collected more than 20 years ago.

“This paper does not provide evidence that PFAS exposure causes aging, nor does it change the extensive body of scientific and regulatory work already underway to understand and manage specific PFAS of potential concern,” Tom Flanagin, the council’s senior director of communications, said in an email.

The study, published Thursday in the journal Frontiers in Aging, used public data from a randomly chosen group of 326 older women and men enrolled in 1999 and 2000 in the US National Health and Nutrition Examination Survey.

Blood samples gathered at the time were examined for the presence of 11 types of PFAS chemicals. The DNA methylome — an epigenetic marker regulating gene expression — was also measured in the blood cells of the participants.

For this study, researchers fed these DNA data into a dozen “epigenetic clocks,” also known as biological clocks, to estimate the aging of blood and other tissues in the participants.

Used since the 1950s to make consumer products nonstick, oil- and water-repellent and resistant to temperature change, PFAS chemicals have been linked to serious health problems, including cancer, fertility issues, high cholesterol, hormone disruption, liver damage, obesity and thyroid disease.

The dangers of so-called legacy PFAS, such as perfluorooctanesulfonic acid, or PFOS, perfluorooctanoic acid, or PFOA, and perfluorohexane sulfonate, or PFHxS, are so well-known they have been targeted for elimination worldwide under the 2001 Stockholm Convention on Persistent Organic Pollutants, a global treaty that would reduce toxic chemicals that bioaccumulate in organisms and the environment. The United States has signed the treaty but has not ratified it.

Under the Biden administration, the US Environmental Protection Agency planned to impose stricter guidelines about legacy PFAS levels, including labeling them as “hazardous substances” under the US Superfund law. However, those plans have been rolled back or delayed under the Trump administration.

The chemical industry has created many other forms of PFAS that have not been as widely studied. Those chemicals may well have the same biological impacts as the rest of the PFAS family — and that’s what the new study found, Li said.

“Our results suggest that some less-studied PFAS — namely perfluorononanoic acid (PFNA) and perfluorooctanesulfonamide (PFOSA) — may also have biologically meaningful associations,” Li said.

In fact, the study found higher concentrations of PFNA and PFOSA were strong predictors of faster epigenetic aging in men between 50 and 64 years of age but not in women.

Despite these findings, “it’s important not to panic,” Li said. “Our study shows associations, not proof of causation. PFAS exposure is widespread, and complete avoidance is unrealistic.

“However, reducing exposure where feasible — such as using certified water filters, following local water advisories, and minimizing contact with stain- or grease-resistant materials — may be reasonable steps,” he added. “At the same time, meaningful risk reduction depends largely on regulatory action and environmental cleanup, since many exposures occur at the community level.”

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